News

Image of the Month


 

Along with their colleague Dr. Steven DeKosky, chair of the neurology department at the University of Pittsburgh, the researchers are currently using the technique in patients with dementia of unknown origin.

PET with PIB reveals differences in amyloid deposition between cognitively normal subjects (far left) and subjects with AD (far right). PET with PIB also reveals the range of amyloid accumulation in subjects with clinically mild cognitive impairment (center). Courtesy Dr. William E. Klunk, Ph.D. and Chester Mathis, Ph.D./University of Pittsburgh

Pages

Recommended Reading

Diet, Exercise May Reduce Alzheimer's Risk in Apo E ϵ4 Carrier
MDedge Neurology
Amyloid Angiopathy Heaviest in Occipital Region
MDedge Neurology
Removal of Amyloid May Increase Neuronal Oxidation
MDedge Neurology
Quantitative EEG Diagnostic of Dementia Subtype
MDedge Neurology
Clamping Changes Reduce Post-CABG Deficits
MDedge Neurology
TBI Test Identifies Those at Risk for Cognitive Decline After Surgery
MDedge Neurology
FDA Approves Changes in Clozapine Blood Monitoring
MDedge Neurology
Sleep Apnea May Be Key to Apo E4 and Alzheimer's Link
MDedge Neurology
Hypertension May Impair Memory
MDedge Neurology
Impaired Graphesthesia May Flag Early Alzheimer's
MDedge Neurology