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Vedolizumab shows promise in Crohn’s, ulcerative colitis

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A new biologic on the block: a little slower but maybe safer

Dr. Siddhartha Parker
Vedolizumab uses gut-selective blockade of the

alpha4beta7 subunit to affect lymphocyte trafficking. In the GEMINI studies,

benefit over placebo was seen in both ulcerative colitis (UC) and Crohn’s

disease. While both primary and secondary endpoints were clinically significant

in UC, the results were not quite as robust for Crohn’s disease, which may be

due to the relatively early timing (6 weeks) of the coprimary endpoint

assessment. Even so, the GEMINI studies show some of the most promising

maintenance data seen for inflammatory bowel disease therapy, in addition to a

low rate of developing antibodies against vedolizumab (4%).

The safety profile is

equally encouraging. Likely due to vedolizumab’s gut-selective blockage,

serious infections may occur less often than with other biologic agents.

Furthermore, its alpha4beta7 selectivity differentiates it from natalizumab,

theoretically eliminating the risk of progressive multifocal

leukoencephalopathy (PML). No cases of PML have been reported in the large drug

development program.

Dr. Corey A. Siegel
With its apparent

durability of response and reassuring safety profile, vedolizumab may in fact

be positioned earlier in the treatment paradigm than other immune-suppressive

agents. At least for UC, it is reasonable to consider its use after

5-aminosalicylates fail. Vedolizumab’s somewhat slower onset when compared to anti–tumor

necrosis factor (anti-TNF) agents may require either patience if symptoms are

tolerable, or the coadministration of corticosteroids to induce remission while

waiting for its maintenance benefit to kick in. We hope to use what we’ve

learned about biologics from 15 years of anti-TNFs to quickly determine how to

best optimize vedolizumab in our clinical practice.

Dr. Siddhartha Parker is a fellow in gastroenterology at

Dartmouth-Hitchcock Medical Center, Lebanon, N.H., and Dr. Corey A. Siegel is associate

professor of medicine at the Geisel School of Medicine at Dartmouth, Hanover,

N.H., and director of the Dartmouth-Hitchcock Inflammatory Bowel Disease Center.

Dr. Siegel serves on the advisory boards for Takeda Pharmaceuticals, Abbvie,

Janssen, and UCB.


 

FROM THE NEW ENGLAND JOURNAL OF MEDICINE

The humanized monoclonal antibody vedolizumab is more effective than placebo for induction and maintenance therapy in both ulcerative colitis and Crohn’s disease, according to two separate randomized, controlled phase III studies: GEMINI 1 and GEMINI 2.

The findings from the double-blind, multinational studies were published in the Aug. 22 issue of the New England Journal of Medicine.

GEMINI 1

In GEMINI 1, the clinical response rates at 6 weeks in 374 patients (cohort 1) with active ulcerative colitis who were randomized to receive either induction therapy with vedolizumab or placebo were 47.1% and 25.5%, respectively (P less than .001). In 521 patients (cohort 2) who received open-label vedolizumab, the clinical response rate at 6 weeks was 44.3%.

In a trial of maintenance therapy, those patients from both cohorts who responded to vedolizumab at week 6 were then randomized to receive either vedolizumab or placebo every 4 or 8 weeks for up to 52 weeks. The clinical remission rates at 52 weeks were 44.8% in the group that received vedolizumab every 4 weeks, 41.8% in the group that received it every 8 weeks, and 15.9% among the patients who were switched to placebo, Dr. Brian G. Feagan of the University of Western Ontario, London, and his colleagues reported on behalf of the GEMINI 1 Study Group.

Patients in the vedolizumab groups were treated with 300 mg IV at weeks 0 and 2. Clinical response was defined as a reduction of at least 3 points in the 0- to 12-point Mayo Clinic score and a decrease of at least 30% from the baseline score, along with a decrease in the rectal bleeding subscore of at least 1 point or an absolute rectal bleeding subscore of 0 or 1. The secondary outcome of clinical remission was defined as a Mayo Clinic score of 2 or less, with no subscore higher than 1, along with mucosal healing, defined by an endoscopic subscore of 0 or 1, the investigators said (N. Engl. J. Med. 2013;369:699-710).

The GEMINI 1 researchers noted that "all prespecified, primary and secondary outcomes in the trial of induction and maintenance therapy were superior in vedolizumab-treated patients versus those who received placebo," and added that longitudinal assessment of a number of factors, such as Mayo Clinic scores and use or dose of glucocorticoids, provided further evidence of a treatment benefit.

Furthermore, disease had been refractory to other treatments in many patients, they noted.

While the study was not designed to identify the time of the maximal effect of treatment as induction therapy, or a minimally effective dose regimen, it appears that treatment every 8 weeks may be an acceptable starting regimen – with dose intensification if needed, they said. "Vedolizumab is effective as both induction and maintenance therapy for patients with moderately to severely active ulcerative colitis," they concluded.

GEMINI 2

In GEMINI 2, the clinical remission rates at 6 weeks in 368 patients with active Crohn’s disease who were randomized to receive either induction therapy with vedolizumab or placebo were 14.5% and 6.8%, respectively (P = .02), and a total of 31.4% and 25.7% of patients, respectively (P = .023), had a Crohn’s Disease Activity Index-100 (CDAI-100) response, defined as a decrease in the CDAI score of at least 100 points. Of 747 patients who received open-label vedolizumab, 17.7% had a clinical remission and 34.4% had a CDAI-100 response at 6 weeks.

Those patients who responded to vedolizumab in the induction phase were randomly assigned to receive either placebo or maintenance treatment every 4 or 8 weeks until week 52. The clinical remission rates at 52 weeks were 36.4% in the group that received the drug every 4 weeks, 39.0% in the group receiving it every 8 weeks, and 21.6% in the placebo group, Dr. William J. Sandborn of the University of California, San Diego, La Jolla, and his colleagues reported on behalf of the GEMINI 2 Study Group (N. Engl. J. Med. 2013; 369:711-21).

As in GEMINI 1, patients in GEMINI 2 who were assigned to the vedolizumab groups were treated with 300 mg IV at weeks 0 and 2. In GEMINI 2, clinical remission was defined as a CDAI score of 150 or less.

In GEMINI 1, the frequency of adverse events was similar in both the treatment and placebo groups, whereas in GEMINI 2, vedolizumab, compared with placebo, was associated with a higher rate of serious adverse events (24.4% vs. 15.3%), infections (44.1% vs. 40.2%), and serious infections (5.5% vs. 3.0%), the investigators said.

Patients in both GEMINI 1 and 2 were aged 18-80 years who were enrolled between 2008 and 2012 through more than 200 participating medical centers in more than 30 countries.

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