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Meta-Analysis Finds No Evidence of Statin-Cancer Link


 

Major Finding: No increased risk for cancer or cancer death occurred in patients who were treated with statins, compared with those treated by placebo or lower statin dosages.

Data Source: Meta-analysis of 26 randomized, controlled trials of statins that each enrolled at least 1,000 patients who were followed for at least 2 years.

Disclosures: Dr. Emberson said he had no disclosures.

STOCKHOLM – Data from 170,000 patients in 26 randomized trials may finally dismiss the idea that low cholesterol levels during statin therapy play a role in causing cancer.

“There is no evidence that cancer risk is increased when very low cholesterol concentrations are achieved with high-dose statins,” Jonathan Emberson, Ph.D., said at the congress.”

“There is no suggestion of an emergence of any hazard with longer duration of treatment, at least within a period of about 5 years. There is no evidence that low cholesterol increases cancer risk at any site or in any group of individuals. I think the question about statins has now probably been answered as well as it can be from the randomized trials,” said Dr. Emberson, a statistician in the clinical trial service unit at the University of Oxford (England).

Follow-up for the patients in the trials ran 5-6 years, producing “extremely reassuring” results for long-term safety, he said in an interview.

Concern that very low serum cholesterol levels – hence statin therapy – might boost cancer incidence has not had a big impact on statin use. But every now and then over the past 20 years, “a trial threw out a random result that raised a new hypothesis,” he said. For example, in 1996, the CARE (Cholesterol and Recurrent Events) trial, which compared 40-mg pravastatin with placebo for secondary prevention in more than 4,000 patients followed for an average of 5 years, showed 12 cases of breast cancer during follow-up in the pravastatin arm, compared with one case in the placebo arm, a statistically significant difference (N. Engl. J. Med. 1996;335:1001-9).

“Random things happen all the time,” Dr. Emberson noted. The CARE results showed “a significant excess, but what's important is, it wasn't supported by data from all the other statin trials. Occasionally, trials throw up hypotheses that can be tested. We attempted to systematically test all of those hypotheses using all of the data.”

The 26 statin trials in the meta-analysis included all those that were published through the end of 2009 with at least 1,000 patients followed for at least 2 years. In all, 21 trials compared a statin with placebo, and 5 compared a low statin dose with a higher statin dose. The 170,000 patients in all 26 trials developed 10,000 cases of cancer during follow-up, with more than 3,500 cancer deaths.

Statin treatment gave no hint of an influence on cancer rates in all 26 studies together, nor separately in the 21 studies in which it was compared with placebo, nor in the 5 in which high dose was compared with low dose. The results showed no sign of cancer increase when treated patients started with low serum levels of LDL cholesterol. There was no cancer impact with longer duration of statin use, no impact for various, specific cancer types, and no difference by age or by sex. The analysis showed no suggestion of an increased risk in the elderly. And no increased risk appeared for gastrointestinal cancers, another cancer type that gave a signal for higher risk in one of the trials.

“The value of our analysis is that we were able to systematically test all of the hypotheses that had been raised in a much larger data set than has previously been possible, and the results are very reassuring for the many millions of patients who take statins, at least for 5-6 years' duration.” In addition, “results from studies that followed patients for longer than 6 years have not suggested any cancer concerns,” Dr. Emberson said.

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